Posted 30 November 2003 - 09:11 PM
Rickoshea,Always good to hear from someone who has studied these issues. Your points are very helpful and I would pay no attention to anyone who finds fault with it.I suspect that certain bacteria would increase plaque buildup over other types. I chose steptococcus as an example because I had read a few things about it. But i agree that many types would cause plaque along with the saliva. you are also correct about the bacteria in the mouth reverting back to its original state. i ususally use the vsl#3 (almost a 1/2 trillion bacteria per packet) and it manages to stay in my mouth because it becomes very gummy. but this bacteria does not implant. it leaves the system after a couple weeks. i do wonder if certain bacteria would be less likely to form plaque due to different metabolic byproducts that are formed???? Ecology of viridans streptococci in the oral cavity and pharynx.Frandsen EV, Pedrazzoli V, Kilian M.Department of Oral Biology, Royal Dental College, Arhus, Denmark.Recently published taxonomic studies of viridans streptococci have resulted in several changes in the nomenclature and definition of oral streptococcal species. With this background, the ecology of streptococci in the oropharyngeal cavities was reinvestigated. The results based on the examination of 1426 streptococcal isolates confirmed and extended earlier findings. Apart from mature supragingival plaque, which contained a mixture of all orally encountered streptococci, each site showed a characteristic streptococcal flora. Initial dental plaque formation is primarily associated with Streptococcus sanguis, Streptococcus mitis biovar 1 and Streptococcus oralis. Our investigation showed that S. sanguis and S. mitis biovar 1 were the most prominent streptococci, also on buccal mucosa. In contrast, S. oralis was almost exclusively found in initial dental plaque. Streptococcus gordonii, formerly part of S. sanguis, was found in small numbers on the oropharyngeal mucosa and in mature supragingival plaque. The dorsum of the tongue was dominated by S. mitis biovar 2 and Streptococcus salivarius, the latter of which was predominant also on the pharyngeal mucosa. Streptococcus anginosus was by far the most predominant streptococcus in subgingival plaque. Immunoglobulin A1 (IgA1) protease-producing streptococci were primarily isolated from initial dental plaque and from the buccal mucosa. This lends further support to the concept of IgA1 proteases being important for the ability of streptococci to evade the local immune defence during their initial colonization of certain oral surfaces. images/Oral Anatomy/HairyTongue.jpgBlack or white hairy tongue--This condition is covered above. It is NOT a sign of incurable disease. It usually occurs during ordinary febrile illnesses and the "hair" can be scraped off easily with a tongue scraper. The hairy coating is a breeding ground for various bacteria and yeasts, and sometimes responds well to topical fungicides such as Nystatin. This condition is not contagious. this is probably why the nystatin made me so incredibly ill when i first started to use it. this sick feeling went away after awhile.
My IBS is gone! (almost 8 months now)
There is Hope!